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dc.contributor.advisorNadel, Lynnen_US
dc.contributor.authorWarren, Stacey Gayle, 1962-
dc.creatorWarren, Stacey Gayle, 1962-en_US
dc.date.accessioned2013-03-28T10:36:53Z
dc.date.available2013-03-28T10:36:53Z
dc.date.issued1990en_US
dc.identifier.urihttp://hdl.handle.net/10150/277316
dc.description.abstractThe effects of postnatal zinc deficiency on the development of the hippocampal formation was assessed using the Morris water tank task. Tests at days 28, 29, 88 and 89 revealed no differences between ad libitum zinc deficient animals and ad libitum controls. Subjects whose food intake was restricted but was zinc adequate were impaired on the hippocampal version of this task but not the control version. These results suggest that previous reports of hippocampal impairment secondary to zinc deficiency should be re-evaluated.
dc.language.isoen_USen_US
dc.publisherThe University of Arizona.en_US
dc.rightsCopyright © is held by the author. Digital access to this material is made possible by the University Libraries, University of Arizona. Further transmission, reproduction or presentation (such as public display or performance) of protected items is prohibited except with permission of the author.en_US
dc.subjectPsychology, Psychobiology.en_US
dc.subjectHealth Sciences, Nutrition.en_US
dc.titleThe effects of postnatal zinc deficiency on spatial learning in ratsen_US
dc.typetexten_US
dc.typeThesis-Reproduction (electronic)en_US
thesis.degree.grantorUniversity of Arizonaen_US
thesis.degree.levelmastersen_US
dc.identifier.proquest1340717en_US
thesis.degree.disciplineGraduate Collegeen_US
thesis.degree.disciplinePsychologyen_US
thesis.degree.nameM.A.en_US
dc.identifier.bibrecord.b26281831en_US
refterms.dateFOA2018-08-27T11:25:26Z
html.description.abstractThe effects of postnatal zinc deficiency on the development of the hippocampal formation was assessed using the Morris water tank task. Tests at days 28, 29, 88 and 89 revealed no differences between ad libitum zinc deficient animals and ad libitum controls. Subjects whose food intake was restricted but was zinc adequate were impaired on the hippocampal version of this task but not the control version. These results suggest that previous reports of hippocampal impairment secondary to zinc deficiency should be re-evaluated.


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