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dc.contributor.authorFadare, Oluwole
dc.contributor.authorZheng, Wenxin
dc.date.accessioned2016-05-20T08:59:15Z
dc.date.available2016-05-20T08:59:15Z
dc.date.issued2008en
dc.identifier.citationDiagnostic Pathology 2008, 3:6 doi:10.1186/1746-1596-3-6en
dc.identifier.doi10.1186/1746-1596-3-6en
dc.identifier.urihttp://hdl.handle.net/10150/610131
dc.description.abstractIn this article, the authors briefly review the historical evolution of the various putative precursor lesions for Type II endometrial cancers, with an emphasis on the newly defined "Endometrial Glandular Dysplasia (EmGD)". The evidentiary basis for delineating serous EmGD as the most probable precursor lesions to endometrial serous carcinoma is reviewed in detail. An argument is advanced for the discontinuation of the term serous "endometrial intraepithelial carcinoma (EIC)" as a descriptor for a supposedly intraepithelial, precancerous lesion. Preliminary evidence is also presented that suggests that there is a morphologically recognizable "clear cell EmGD" that probably represents a precancerous lesion to endometrial clear cell carcinomas.
dc.language.isoenen
dc.publisherBioMed Centralen
dc.relation.urlhttp://www.diagnosticpathology.org/content/3/1/6en
dc.rights© 2008 Fadare and Zheng; licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0).en
dc.rights.urihttps://creativecommons.org/licenses/by/2.0/
dc.titleEndometrial Glandular Dysplasia (EmGD): morphologically and biologically distinctive putative precursor lesions of Type II endometrial cancersen
dc.typeArticleen
dc.identifier.eissn1746-1596en
dc.contributor.departmentDepartment of Pathology, Wilford Hall Medical Center, Lackland Air Force Base, San Antonio, Texas, USAen
dc.contributor.departmentDepartment of Pathology, University of Texas Health Science Center at San Antonio, San Antonio, Texas, USAen
dc.contributor.departmentDepartment of Pathology, Department of Obstetrics and Gynecology, and the Arizona Cancer Center, University of Arizona College of Medicine, Tucson, Arizona, USAen
dc.contributor.departmentDepartment of Pathology and Hospital of Obstetrics and Gynecology, Shanghai Medical College, Fudan University, Shanghai, Chinaen
dc.identifier.journalDiagnostic Pathologyen
dc.description.collectioninformationThis item is part of the UA Faculty Publications collection. For more information this item or other items in the UA Campus Repository, contact the University of Arizona Libraries at repository@u.library.arizona.edu.en
dc.eprint.versionFinal published versionen
refterms.dateFOA2018-09-11T10:48:57Z
html.description.abstractIn this article, the authors briefly review the historical evolution of the various putative precursor lesions for Type II endometrial cancers, with an emphasis on the newly defined "Endometrial Glandular Dysplasia (EmGD)". The evidentiary basis for delineating serous EmGD as the most probable precursor lesions to endometrial serous carcinoma is reviewed in detail. An argument is advanced for the discontinuation of the term serous "endometrial intraepithelial carcinoma (EIC)" as a descriptor for a supposedly intraepithelial, precancerous lesion. Preliminary evidence is also presented that suggests that there is a morphologically recognizable "clear cell EmGD" that probably represents a precancerous lesion to endometrial clear cell carcinomas.


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© 2008 Fadare and Zheng; licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0).
Except where otherwise noted, this item's license is described as © 2008 Fadare and Zheng; licensee BioMed Central Ltd. This is an Open Access article distributed under the terms of the Creative Commons Attribution License (http://creativecommons.org/licenses/by/2.0).