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dc.contributor.authorAskari-Khorasgani, Omid
dc.contributor.authorPessarakli, Mohammad
dc.date.accessioned2019-04-25T23:24:16Z
dc.date.available2019-04-25T23:24:16Z
dc.date.issued2019
dc.identifier.citationAskari-Khorasgani, O., & Pessarakli, M. (2019). Manipulation of plant methylglyoxal metabolic and signaling pathways for improving tolerance to drought stress. Journal of Plant Nutrition, 1-8.en_US
dc.identifier.issn0190-4167
dc.identifier.doi10.1080/01904167.2019.1589502
dc.identifier.urihttp://hdl.handle.net/10150/632111
dc.description.abstractDrought stress is considered one of the major constraints to crop production with devastating effects worldwide. Methylglyoxal (MG) homeostasis plays an essential role in promoting plant growth, development, metabolic adaptation, signal transduction, and thereby responses to drought stress. Manipulation of genes encoding aldose-aldehyde reductases (ALRs), aldo– keto reductases (AKRs), genes that are involved in maintaining high ascorbate:glutathione (AsA:GSH), glutathione:glutathione disulfide (GSH:GSSG) contents and ratios, as well as jasmonic acid (JA) biosynthesis are of critical importance for improving MG detoxification, and thereby tolerance to drought stress. MG detoxification by maintaining optimal abscisic acid (ABA), JA, salicyclic acid (SA), and brassinosteroid (BR) homeostasis and crosstalk is also essential to optimize plant performance under both normal and drought stress conditions.en_US
dc.language.isoenen_US
dc.publisherTAYLOR & FRANCIS INCen_US
dc.rights© 2019 Taylor & Francis Group, LLCen_US
dc.subjectantioxidanten_US
dc.subjecthormonehomeostasisen_US
dc.subjectmethylglyoxaldetoxificationen_US
dc.subjectredoxen_US
dc.subjectstresssignalen_US
dc.subjectwater stressen_US
dc.titleManipulation of plant methylglyoxal metabolic and signaling pathways for improving tolerance to drought stressen_US
dc.typeArticleen_US
dc.contributor.departmentUniv Arizona, Sch Plant Scien_US
dc.identifier.journalJournal of Plant Nutritionen_US
dc.description.note12 month embargo; published online: 07 Apr 2019en_US
dc.description.collectioninformationThis item from the UA Faculty Publications collection is made available by the University of Arizona with support from the University of Arizona Libraries. If you have questions, please contact us at repository@u.library.arizona.edu.en_US
dc.eprint.versionFinal accepted manuscripten_US


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